Investigators publish new data in the report 'Cyclophilin B as a co-regulator of prolactin-induced gene expression and function in breast cancer cells.' According to a study from the United States, The effects of prolactin (PRL) during the pathogenesis of breast cancer are mediated in part though Stat5 activity enhanced by its interaction with its transcriptional inducer, the prolyl isomerase cyclophilin B (CypB).















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